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Ros" "autores" => array:1 [ 0 => array:2 [ "nombre" => "E." "apellidos" => "Ros" ] ] ] ] ] "idiomaDefecto" => "es" "EPUB" => "https://multimedia.elsevier.es/PublicationsMultimediaV1/item/epub/S1889183708717252?idApp=UINPBA00004N" "url" => "/18891837/0000002500000001/v1_201307261641/S1889183708717252/v1_201307261641/es/main.assets" ] "itemAnterior" => array:18 [ "pii" => "S1889183708717239" "issn" => "18891837" "doi" => "10.1016/S1889-1837(08)71723-9" "estado" => "S300" "fechaPublicacion" => "2008-01-01" "aid" => "71723" "copyright" => "Sociedad Española de Hipertension-Liga Española para la Lucha de la Hipertensión Arterial (SEH-LELHA)" "documento" => "simple-article" "crossmark" => 0 "subdocumento" => "edi" "cita" => "Hipertens Riesgo Vasc. 2008;25:1-3" "abierto" => array:3 [ "ES" => false "ES2" => false "LATM" => false ] "gratuito" => false "lecturas" => array:2 [ "total" => 2722 "formatos" => array:3 [ "EPUB" => 9 "HTML" => 2436 "PDF" => 277 ] ] "es" => array:7 [ "idiomaDefecto" => true "titulo" => "La hipertensión arterial, ¿un estado de hipercoagulabilidad latente?" "tienePdf" => "es" "tieneTextoCompleto" => 0 "paginas" => array:1 [ 0 => array:2 [ "paginaInicial" => "1" "paginaFinal" => "3" ] ] "contienePdf" => array:1 [ "es" => true ] "autores" => array:1 [ 0 => array:2 [ "autoresLista" => "V. Barrios Alonso, A. Calderón Montero, C. Escobar Cervantes" "autores" => array:3 [ 0 => array:2 [ "nombre" => "V." "apellidos" => "Barrios Alonso" ] 1 => array:2 [ "nombre" => "A." "apellidos" => "Calderón Montero" ] 2 => array:2 [ "nombre" => "C." "apellidos" => "Escobar Cervantes" ] ] ] ] ] "idiomaDefecto" => "es" "EPUB" => "https://multimedia.elsevier.es/PublicationsMultimediaV1/item/epub/S1889183708717239?idApp=UINPBA00004N" "url" => "/18891837/0000002500000001/v1_201307261641/S1889183708717239/v1_201307261641/es/main.assets" ] "es" => array:12 [ "idiomaDefecto" => true "titulo" => "Hipertensión arterial, hipercoagulabilidad y cardiopatía isquémica" "tieneTextoCompleto" => 0 "paginas" => array:1 [ 0 => array:2 [ "paginaInicial" => "4" "paginaFinal" => "8" ] ] "autores" => array:1 [ 0 => array:4 [ "autoresLista" => "B. Virgós-Señor, A.C. Nebra-Puertas, M.A. Suárez-Pinilla, R. Cornudella-Lacasa, P. Portero-Pérez" "autores" => array:5 [ 0 => array:4 [ "nombre" => "B." "apellidos" => "Virgós-Señor" "email" => array:1 [ 0 => "s976358224@wanadoo.es" ] "referencia" => array:2 [ 0 => array:2 [ "etiqueta" => "<span class="elsevierStyleSup">a</span>" "identificador" => "aff0005" ] 1 => array:2 [ "etiqueta" => "<span class="elsevierStyleSup">†</span>" "identificador" => "cor0005" ] ] ] 1 => array:3 [ "nombre" => "A.C." "apellidos" => "Nebra-Puertas" "referencia" => array:1 [ 0 => array:2 [ "etiqueta" => "<span class="elsevierStyleSup">a</span>" "identificador" => "aff0005" ] ] ] 2 => array:3 [ "nombre" => "M.A." "apellidos" => "Suárez-Pinilla" "referencia" => array:1 [ 0 => array:2 [ "etiqueta" => "<span class="elsevierStyleSup">b</span>" "identificador" => "aff0010" ] ] ] 3 => array:3 [ "nombre" => "R." "apellidos" => "Cornudella-Lacasa" "referencia" => array:1 [ 0 => array:2 [ "etiqueta" => "<span class="elsevierStyleSup">c</span>" "identificador" => "aff0015" ] ] ] 4 => array:3 [ "nombre" => "P." "apellidos" => "Portero-Pérez" "referencia" => array:1 [ 0 => array:2 [ "etiqueta" => "<span class="elsevierStyleSup">d</span>" "identificador" => "aff0020" ] ] ] ] "afiliaciones" => array:4 [ 0 => array:3 [ "entidad" => "Servicio de Medicina Intensiva. Hospital Universitario Miguel Servet. Zaragoza. España" "etiqueta" => "<span class="elsevierStyleSup">a</span>" "identificador" => "aff0005" ] 1 => array:3 [ "entidad" => "Servicios de Medicina Intensiva" "etiqueta" => "<span class="elsevierStyleSup">b</span>" "identificador" => "aff0010" ] 2 => array:3 [ "entidad" => "Hematología" "etiqueta" => "<span class="elsevierStyleSup">c</span>" "identificador" => "aff0015" ] 3 => array:3 [ "entidad" => "Cardiología. Hospital Clínico. Zaragoza. España" "etiqueta" => "<span class="elsevierStyleSup">d</span>" "identificador" => "aff0020" ] ] "correspondencia" => array:1 [ 0 => array:3 [ "identificador" => "cor0005" "etiqueta" => "†" "correspondencia" => "Correspondencia: Av. de Valencia, 51-53, esc. 1.a, piso 8.o B. 50005 Zaragoza. España." ] ] ] ] "titulosAlternativos" => array:1 [ "en" => array:1 [ "titulo" => "Arterial hypertension, hypercoagulability and ischemic heart disease" ] ] "pdfFichero" => "main.pdf" "tienePdf" => true "PalabrasClave" => array:2 [ "es" => array:1 [ 0 => array:4 [ "clase" => "keyword" "titulo" => "Palabras clave" "identificador" => "xpalclavsec219046" "palabras" => array:4 [ 0 => "hipertensión arterial" 1 => "cardiopatía isquémica" 2 => "fragmento 1+2 de la protrombina" 3 => "complejo trombina-antitrombina" ] ] ] "en" => array:1 [ 0 => array:4 [ "clase" => "keyword" "titulo" => "Key words" "identificador" => "xpalclavsec219045" "palabras" => array:4 [ 0 => "arterial hypertension" 1 => "ischemic heart disease" 2 => "fragment 1+2 of prothrombin" 3 => "thrombin-antithrombin complex" ] ] ] ] "tieneResumen" => true "resumen" => array:2 [ "es" => array:1 [ "resumen" => "<span class="elsevierStyleSectionTitle">Introducción</span><p id="spar0005" class="elsevierStyleSimplePara elsevierViewall">La enfermedad ateromatosa coronaria es un proceso crónico favorecido por los factores de riesgo cardiovascular, entre los que está la hipertensión arterial (HTA), y caracterizado tanto por la existencia de fenómenos inflamatorios como por la presencia de un activación subclínica de la coagulación.</p> <span class="elsevierStyleSectionTitle">Pacientes y métodos</span><p id="spar0010" class="elsevierStyleSimplePara elsevierViewall">Estudio observacional de casos y controles sobre 40 pacientes hipertensos sin más factores de riesgo cardiovascular, con enfermedad coronaria y coronariografía. Realizamos un grupo control de 15 sujetos sanos sin factores de riesgo cardiovascular. Comparamos el estudio estándar de coagulación y la actividad subclínica de la hemostasia sanguínea midiendo el fragmento 1+2 de la protrombina (F1+2) y el complejo trombina-antitrombina (TAT), entre ambos grupos.</p> <span class="elsevierStyleSectionTitle">Resultados</span><p id="spar0015" class="elsevierStyleSimplePara elsevierViewall">Los pacientes con HTA y cardiopatía isquémica, en los que la coronariografía evidencia una lesión obstructiva, presentan unos niveles más elevados tanto de F1+2, como de factor de TAT que las personas sanas. Estos niveles pueden verse modificados por el tratamiento antihipertensivo seguido.</p> <span class="elsevierStyleSectionTitle">Conclusiones</span><p id="spar0020" class="elsevierStyleSimplePara elsevierViewall">La HTA promueve un estado de hipercoagulabilidad subclínico, relacionado con la fisiopatología de la cardiopatía isquémica, que podría verse modificado por el tratamiento seguido por los pacientes.</p>" ] "en" => array:1 [ "resumen" => "<span class="elsevierStyleSectionTitle">Introduction</span><p id="spar0025" class="elsevierStyleSimplePara elsevierViewall">Coronary artery disease is a chronic condition favored by cardiovascular risk factors, among which arterial hypertension is found. It is characterized by the existence of inflammatory phenomena as well as by the presence of subclinical activation of coagulation.</p> <span class="elsevierStyleSectionTitle">Patients and methods</span><p id="spar0030" class="elsevierStyleSimplePara elsevierViewall">Observational case-control study on 40 hypertense patients without any other cardiovascular risk factors, with coronary disease demonstrated by coronary angiography. There was a control group of 15 healthy subjects with no cardiovascular risk factors. We compared the standard coagulation study and subclinical activity of hemostasis, measuring fragment 1+2 of prothrombin (F1+2) and the thrombin-antithrombin complex (TAT) between both groups.</p> <span class="elsevierStyleSectionTitle">Results</span><p id="spar0035" class="elsevierStyleSimplePara elsevierViewall">Patients with arterial hypertension and ischemic heart disease, in whom the coronariography showed an obstructive lesion, had higher levels of both F1+2 and TAT factor than healthy people. These levels could be modified with antihypertensive treatment.</p> <span class="elsevierStyleSectionTitle">Conclusions</span><p id="spar0040" class="elsevierStyleSimplePara elsevierViewall">Arterial hypertension is associated with a subclinical hypercoagulability that is related with the pathophysiology of ischemic heart disease. 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Journal Information
Hipertensión arterial, hipercoagulabilidad y cardiopatía isquémica
Arterial hypertension, hypercoagulability and ischemic heart disease