Información del artículo
El Texto completo está disponible en PDF
REFERENCIAS
[1.]
A. Walker, G. Mashour.
A brief history of anesthesia and sleep.
International anesthesiology clinics., 46 (2008), pp. 1-10
[2.]
G.A. Mashour, S.A. Forman, J.A. Campagna.
Mechanism of anesthesia general, from molecules to mind.
Best Practice & Research Clinical Anaesthesiology, 19 (2006), pp. 349-364
[3.]
R. Uwe, A. Bernd.
Molecular and neuronal substrates for general anesthetics.
Nature reviews of neuroscience, volume 4 (2004), pp. 709-716
[4.]
A. Bruce, D. Bray.
Alberts Molecular Biology of the cell.
Fourth edition, Mc Graw Hill, (2007),
[5.]
K.W. Miller.
The natural of sites action of anesthetics.
British journal of anaesthesia, 89 (2002), pp. 17-21
[6.]
H.A. Nasch.
In vivo genetics of anesthetic actions.
British journal of anaesthesia, 89 (2002), pp. 143-155
[7.]
L.J. Velly, M.F. Rey, N.J. Bruder, F.A. Gouvitsos.
The search for structure and mechanisms controlling anesthesia induced unconsciousness.
Anesthesiology, 107 (2007), pp. 105-110
[8.]
J.R. Atack.
Anxioselective Compounds Acting at the GABAA Receptor Benzodiazepine Binding Site.
Current Drug Targets - CNS & Neurological Disorders, 24 (2003), pp. 213-232
[9.]
E. Moody, P. Skolnick.
Book of Molecular Basis of anesthesia.
CRC Editors, University, (2001),
[10.]
J.F. Antognini, E. Carstens.
In vivo characterization of clinical anesthesia and components.
British journal of anaesthesia., 89 (2002), pp. 156-166
[11.]
K. Solt, S.A. Forman.
Correlating the clinical actions and molecular mechanisms of general anesthetics.
Current Opinion in Anesthesiology., 20 (2007), pp. 300-306
[12.]
J.R. Trudell, E. Bertaccili.
molecular modeling of specific and non specific anesthetic interaction.
British journal of anaesthesia., 89 (2002), pp. 32-40
[13.]
B.W. Urban.
Current assessments of targets and theories of anesthesia.
British journal of anaesthesia., 167 (2002), pp. 83-89
[14.]
B.W. Urban, M. Bleckwen.
Concepts and correlations relevant to general anesthesia.
British journal of anaesthesia., 89 (2002), pp. 8-16
[15.]
Y. Zhang, Michael, M.J. Laster, C.R. Stabernack, J.M. Sonner.
Blockade of 5-HT2A Receptors May Mediate or Modulate Part of the Immobility Produced by Inhaled Anesthetics.
Anesthesia Analgesia, 97 (2003), pp. 475-479
[16.]
J.L. Steiger, Sh. Russek.
Available Pharmacology and therapeutics GABAa receptors: building the bridge between subunit mRNAm promoters, and cognate transcription factors.
Anesthesia Analgesia, 101 (2004), pp. 261-262
[17.]
C. Grasshoft, R. Uwe, A. Bernd.
Molecular and mechanisms of general anesthesia the multi site and multiple mechanisms concept.
Current opinion in anaesthesiology., 18 (2005), pp. 386-391
[18.]
A.J. Smith, P.B. Simpson.
Methodological approaches for the study of GABAa receptor pharmacology and functional responses Analytical & Bioanalytical Chemistry..
Springer Berlin / Heildelberg, 77 (2003), pp. 843-851
[19.]
N.G. Bowery, T.G. Smart.
GABA and glycine as neurotransmitters: a brief history.
British Journal of Pharmacology, 47 (2006), pp. S109-S119
[20.]
M.J. Rebechii, S.N. Pentyala.
Anaesthetic actions on other targets: protein kinase C and guanine nucleotide binding proteins.
British journal of anesthesia., 89 (2002), pp. 62-78
[21.]
Y. Zhang, J.M. Sonner, E.I. Eger.
Gamma-Aminobutyric Acid A Receptors Do Not Mediate the Immobility Produced by Isoflurane.
Anesthesia Analgesia., 99 (2004), pp. 85-90
[22.]
P.J. Whiting.
GABA-A receptor subtypes in the brain: a paradigm for CNS drug discovery?.
Drug discovery today., 8 (2003), pp. 445-450
[23.]
J.P. Dilger.
The effects of general anaesthetics on ligated ion channels.
British Journal of Anaesthesia., 89 (2002), pp. 41-51
[24.]
P. Flood, K. Matheu.
Intravenous anesthetic differentially modulates ligand ion chanels.
Anesthesiology, (2000), pp. 1418-1425
[25.]
Y. Zhang, M.J. Laster, K. Hara, R.A. Harris, E.I. Eger.
Glycine receptors mediate part of the immobility produced by inhaled anesthetics.
Anesthesia and Analgesia., 96 (2003), pp. 97-101
[26.]
P. Flood, K. Matthew.
Intravenous Anesthetics Differentially Modulate Ligand-gated Ion Channels.
Anesthesiology., 92 (2000), pp. 1418-1422
[27.]
C. Grasshoff, B. Drexler1, R. Uwe, A. Bernd.
Anaesthetic Drugs: Linking Molecular Actions to Clinical Effects.
Current Pharmaceutical Design., 12 (2006), pp. 3665-3679
[28.]
R. Miramontes.
Relación de canales de potasio acoplados a proteinas G y fármacos estado actual y perspectivas, Tesis maestria en Ciencias fisiológicas, Universidad de Colina Mexico.
[29.]
C. Heurteaux1, N. Guy1, C. Laigle, N. Blondeau.
TREK-1, a KÞ channel involved in neuroprotection and general anesthesia.
EMBO Journal, 23 (2004), pp. 2684-2695
[30.]
W. Heinke.
In vivo image of anesthetic action in humans: approaches with positron emission tomography (PET) and functional magnetic resonance imaging.
British journal of anaesthesia., 89 (2002), pp. 112-122
[31.]
E. Tassonyi, E. Charpantier.
The role of nicotinic acetylcholine receptors in the mechanisms of anesthesia.
Brain Research Bulletin., 57 (2002), pp. 133-150
[32.]
B. Anthowiak.
In vitro networks: cortical mechanisms of anesthetic action.
British journal of anaesthesia., 89 (2002), pp. 156-166
[33.]
J.J. Kending.
In vitro networks: subcortical mechanisms of anesthetic action.
British journal of anaesthesia., 89 (2002), pp. 91-104
[34.]
C.D. Richards.
Anaesthetic modulation of synaptic transmission in the mammalian CNS.
British journal of anaesthesia., 89 (2002), pp. 79-90
[35.]
A. Zeller, M. Arras, R. Juran, R. Uwe.
Identification of a molecular target mediating the general anesthetic actions of pentobarbital.
Mol Pharmacol., 71 (2007), pp. 852-859
[36.]
Esquema receptor GABA.
Modificado de de.
[37.]
Original de Nature Reviews Neuroscience, 6 (July 2005), pp. 565-575
Copyright © 2009. Revista Colombiana de Anestesiología
Opciones de artículo