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Vol. 34. Issue S1.
Jornada de Actualización en Gastroenterología Aplicada
Pages 43-46 (January 2011)
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Vol. 34. Issue S1.
Jornada de Actualización en Gastroenterología Aplicada
Pages 43-46 (January 2011)
Enfermedad inflamatoria intestinal
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Avances en epidemiología, clínica y diagnóstico de la enfermedad inflamatoria intestinal
Advances in the epidemiology, diagnosis and management of irritable bowel disease
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Miquel Sans
Servicio de Gastroenterología, Hospital Clínic i Provincial/IDIBAPS, CIBER EHD, Barcelona, España
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Resumen

Un año más, la DDW ha sido el foro en el que se han presentado numerosos estudios relacionados con la enfermedad inflamatoria intestinal (EII). Entre ellos destacan numerosos estudios que contribuirán a conocer mejor los factores epidemiológicos que participan en el desarrollo de la enfermedad de Crohn (EC) y la colitis ulcerosa (CU), a caracterizar las infecciones que algunos pacientes presentan, debido al uso de fármacos que alteran la función del sistema inmune, y a mejorar el diagnóstico y la evaluación de la extensión y grado de actividad de la EII mediante técnicas endoscópicas y de imagen. Como en anteriores ediciones, es difícil destacar un único estudio por encima de los demás. De hecho, la relevancia de esta reunión internacional, la más importante en el campo de la gastroenterología, se debe al gran número de contribuciones que suponen un pequeño avance, modesto, pero mejora al fin y al cabo, en el manejo de nuestros pacientes con EC y CU.

Palabras clave:
Enfermedad inflamatoria intestinal
Enfermedad de Crohn
Colitis ulcerosa
Epidemiología
Genética
Clínica
Abstract

One year more, Digestive Disease Week has served as a forum for the presentation of numerous studies on inflammatory bowel disease (IBD). Notable among these studies were several that contributed to a better knowledge of the epidemiological factors that play a role in the development of Crohn's disease (CD) and ulcerative colitis (UC). Studies were also presented that characterized infections in some patients due to the use of drugs that alter immune system function, while others aimed to improve the diagnosis and assessment of IBD extension and activity through endoscopic and imaging techniques.

As in previous years, it is difficult to single out any one particular study. Indeed, the importance of this international meeting, the most important in the field of gastroenterology, lies in the large number of contributions representing a small advance, which although modest, improve the management of patients with CD and UC.

Keywords:
Inflammatory bowel disease
Crohn's disease
Ulcerative colitis
Epidemiology
Genetics
Clinical
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Bibliografía
[1.]
L. Henckaerts, C. Figueroa, S. Vermeire, M. Sans.
The role of genetics in inflammatory bowel disease.
Curr Drug Targets, 9 (2008), pp. 361-368
[2.]
M. Sans, A. Castells.
Ulcerative colitis and Crohn's disease genetics: more similar than we thought?.
Gastroenterology, 135 (2008), pp. 1796-1798
[3.]
M. Parkes, D.P. McGovern, A. Franke, S. Vermeire, E. Louis, T. Ahmad, et al.
18 New Crohn's disease susceptibility genes and loci identified by the international IBD Genetics Consortium.
[4.]
S.Y. Shaw, J. Blanchard, C.N. Bernstein.
Antibiotic prescriptions in the first year of life increases the risk of pediatric inflammatory bowel disease: a population-based analysis.
[5.]
S.S. Chan.
Aspirin in the aetiology of Crohn's disease and ulcerative colitis – results from a European prospective cohort study.
[6.]
A.N. Ananthakrishnan, E.L. McGinley, D.G. Binion, K. Saeian.
Air pollution and hospitalizations for inflammatory bowel disease: an ecologic analysis.
[7.]
L.A. García Rodríguez, A. Ruigómez, J. Panés.
Acute gastroenteritis is followed by an increased risk of inflammatory bowel disease.
Gastroenterology, 130 (2006), pp. 1588-1594
[8.]
A. Ruigómez, L.A. García Rodríguez, J. Panés.
Risk of irritable bowel syndrome after an episode of bacterial gastroenteritis in general practice: influence of comorbidities.
Clin Gastroenterol Hepatol, 5 (2007), pp. 465-469
[9.]
F. Mearin, M. Pérez-Oliveras, A. Perelló, J. Vinyet, A. Ibáñez, J. Coderch, et al.
Dyspepsia and irritable bowel syndrome after a Salmonella gastroenteritis outbreak: one-year follow-up cohort study.
Gastroenterology, 129 (2005), pp. 98-104
[10.]
F. Mearin, A. Perelló, A. Balboa, M. Perona, M. Sans, A. Salas, et al.
Pathogenic mechanisms of postinfectious functional gastrointestinal disorders: results 3 years after gastroenteritis.
Scand J Gastroenterol, 44 (2009), pp. 1173-1185
[11.]
J.K. Marshall, M. Thabane, A.X. Garg, W.F. Clark, P. Moayyedi, S.M. Collins.
Incidence of inflammatory bowel disease following the walkerton outbreak of waterborne bacterial gastroenteritis.
[12.]
P. Jantchou, S. Morois, F. Clavel-Chapelon, M.C. Boutron-Ruault, F. Carbonnel.
Animal protein intake and risk of inflammatory bowel disease: the E3N prospective study.
[13.]
A. Hart.
Total dietary sugar intake and the aetiology of Crohn's disease – data from a European prospective cohort study (EPIC).
[14.]
P.S. De Silva, R. Luben, S.S. Shrestha, A. Welch, K.T. Khaw, A. Hart.
Dietary oleic acid protects against the development of ulcerative colitis – a UK prospective cohort study using data from food diaries.
[15.]
N. Nguyen, K. Bradford, X. Zhang, P. Simpson, S.R. Targan, E.A. Vasiliauskas, et al.
Severity of cytomegalovirus infection on pathology specimens is associated with colectomy rates andresponse to antiviral therapy in inflammatory bowel disease patients.
[16.]
A.N. Ananthakrishnan, E.L. McGinley, K. Saeian, D.G. Binion.
Clostridium difficile and inflammatory bowel disease: has the disease changed in severity from 1998 to 2007?.
[17.]
C.D. Stone, N.A. Accortt, S.H. Magowan.
Prevalence of Clostridium difficile infection in patients with inflammatory bowel disease is significantly related with treatment modality.
[18.]
A.S. Tarnawski, E. Coron, Z. Sandor, J.F. Mosnier, A. Ahluwalia, M. Le Rhun, et al.
Confocal endomicroscopy and molecular imaging demonstrates in colonic mucosa of patients with IBD in remission impaired crypt regeneration, persistant inflammation and pathological angiogenesis. Underlying mechanisms include dysregulation of survivin and aberrant activation of VEGF gene.
[19.]
J. Rimola, S. Rodríguez, O. García-Bosch, I. Ordás, E. Ayala, M. Aceituno, et al.
Magnetic resonance for assessment of disease activity and severity in ileocolonic Crohn's disease.
Gut, 58 (2009), pp. 1113-1120
[20.]
I. Ordás, O. García-Bosch, S. Rodríguez, M. Aceituno, M. Pellise, E. Ricart, et al.
Validation of a magnetic resonance index of activity for ileocolonic Crohn's disease.
Copyright © 2011. Elsevier España S.L.. Todos los derechos reservados
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